Inspiratory muscle weakness is a known consequence of prolonged mechanical ventilation, and there is emerging evidence that specific IMT can ameliorate this weakness. Inspiratory muscle weakness may induce several impairments in both healthy and athletic individuals. Similarly, studies have demonstrated that inspiratory muscle strength also has an important role in the pathophysiology of exercise limitation in several clinical conditions. Exercise limitation is a cardinal manifestation of cardiovascular diseases, and an increasing degree of exercise intolerance is associated with poor prognosis. More recently, studies have found that maximal inspiratory pressure (MIP) is strongly correlated with V’O2peak in patients after acute myocardial infarction and heart failure, reinforcing the influence of the inspiratory muscles on functional capacity.
Author(s) Details:
Rafael Pena,
Graduate Program in Human Movement and Rehabilitation, Evangelical University of Goias, Brazil.
Francisco V. Santos,
Cancer Institute of São Paulo, Intensive Care Unit, São Paulo, Brazil.
Graziella F. B. Cipriano,
Physical Therapy Department, University of Brasilia, Brasilia, Brazil.
Gerson Cipriano Jr.,
Physical Therapy Department, University of Brasilia, Brasilia, Brazil.
Adriana M. Güntzel Chiappa,
Hospital de Clinicas de Porto Alegre, Porto Alegre, Brazil.
Lawrence Patrick Cahalin,
Department of Physical Therapy, University of Miami Miller School of Medicine, Coral Gables, Florida, USA.
Gaspar R. Chiappa,
Graduate Program in Human Movement and Rehabilitation, Evangelical University of Goias, Brazil.
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